Such findings indicate that the aberrant activation of eIF4E, that is mainly observed in HPV‐positive cervical cancer cells, depends on the upregulation of the PI3K/AKT and MEK/ERK pathways on downstream effectors (4EBP1 and MNK1), thereby affecting the rate of translation of CCND1 and ODC1 mRNA. Here, EIF4E is linked to cervical cancer.