MYH11 and CAPN5-related vitreoretinopathy: We discover after chemical injury to the murine eye that lineage-labeled Myh11+ vSMCs-PCs are capable of disembarking from the underlying injured retinal vasculature and phenotypically switching to myofibroblasts that produce copious collagen, a sine qua non of proliferative vitreoretinopathy (PVR)39,40.