Interestingly, a potential role of CD36 in regulating hepatic VLDL secretion was proposed by Nassir et al.82 as they observed that Cd36 deletion in ob/ob mice exacerbated liver steatosis mainly due to reduction in hepatic secretion of VLDL-TGs, ApoB48 and ApoB100. The gene discussed is APOB; the disease is fatty liver disease.