In the present study, we observed a significant increase in CXCL-8, CCL-5, CXCL-9, CCL-2, CXCL-10, and IL-6 among the groups exposed to the venom (NF and HF), previously to antivenom therapy (T0), when compared to the control group (CG). The gene discussed is CXCL9; the disease is hydrops fetalis.