Similarly, emodin can regulate the inflammatory process in experimental autoimmune myocarditis (EAM) (50 mg/kg) and viral myocarditis (VMC) (0.3 mg) model mice by inhibiting the transcription factor NF‐κB, reducing the production of pro-inflammatory cytokines, such as interleukin-1β (IL-1β), interleukin-6 (IL-6), TNF-α, and decreasing the severity of myocarditis (Song et al., 2012; Jiang et al., 2014). Here, IL6 is linked to autoimmune myocarditis.