revealed that FOXD3-AS1 expression was increased in oxygen-glucose deprivation and reoxygenation (OGD/R)-treated H9c2 cells and overexpression of FOXD3-AS1 aggravated ischemia/reperfusion injury of cardiomyocytes through promoting autophagy (Tong et al., 2019). The gene discussed is FOXD3; the disease is ischemia.