While IL-2, CXCL10, and TNF-α contribute to the pathogenesis of sepsis, IL-7, IL-10, GCSF, MCP1, M1P1A are likely elevated as an endogenous attempt to combat sepsis (Snydman et al., 1990; Latifi et al., 2002; Takahashi et al., 2002; Schefold, 2011; Gaballa et al., 2012; Gomes et al., 2013; Herzig et al., 2014; Francois et al., 2018). Here, CCL2 is linked to Sepsis.