Following TGF‐β blockade, tumor‐associated CD11b+Ly6G+neutrophils increased rather than Ly6G− macrophages, and these intratumoral neutrophils play a significant role in tumor cytotoxicity of myeloid cells in anti‐TGF‐β treatment in a CD8+‐dependent manner.36 The gene discussed is ITGAM; the disease is neoplasm.