It was recently reported that postnatal treatment of male Sprague–Dawley rats with BPA (1 mg/kg/day, 21–24 to 49–52 day) resulted in significant neurocognitive disorder in synaptic plasticity and spatial memory and reduced NMDA receptor 2A (NR2A) and AMPA receptor subunit GluR1 expression contributing to memory deficits (75). The gene discussed is GRIA1; the disease is memory.