miR-30c was also suggested to participate in improvement of atherosclerosis and hyperlipidemia via decreasing lipid biosynthesis and secretion of lipoproteins by downregulating lysophosphatidylglycerol acyltransferase 1 (LPGAT1) and microsomal triglyceride transfer protein (MTP) [336]. The gene discussed is LPGAT1; the disease is hyperlipidemia.