SLC1A3 and Esophageal atresia: Thus far, all reported cases have been associated with mutations in SLC1A3 (Jen et al., 2005; de Vries et al., 2009; Pyle et al., 2015; Choi et al., 2017a, b; Iwama et al., 2018), but how these mutations cause the specific clinical phenotype of EA is still insufficiently understood.