The tumor from the left tonsil had integration of HPV16 L2 into intron 6 of the cellular gene CD36. Two blocks from the right tonsil were analyzed; the tumor cells from the right tonsil had integrations of HPV16 E5 into intron 5 of CD36, as well as an additional integration of HPV16 E5 into LAMA3, which likely caused a rearrangement of LAMA3 intron 1 and intron 68. Here, LAMA3 is linked to neoplasm.