Only one animal died after infection with the highest dose assayed of ECTV-mSlfn2 (106 pfu), while the rest of animals survived the disease (Fig. 3C and table S1), indicating that mSlfn1 and mSlfn2 do not complement vSlfn during ECTV infection and suggesting that the strong attenuation observed is mostly due to activation of the cGAS-STING axis in the absence of the p26 domain. The gene discussed is CGAS; the disease is infection.