CP and Wilson disease: This was exemplarily documented in a study investigating a 27-year-old male WD patient, in which the elevated copper concentrations were associated with cerebral iron deposits in treatment-naïve Wilson disease patients, most likely caused by sequestration from dying cells, influx of iron-laden macrophages, increased uptake due to cellular energy production failure, or impaired tissue iron efflux associated with ceruloplasmin dysfunction [36,37].