They predominantly flank the loci harbouring FOXMIND-FOXA1 (Fig. 3) as FOXA1 and FOXMIND are co-duplicated in 89% of the tumours with tandem duplications, driving aberrant FOXA1 and MIPOL1 mRNA expression (Parolia et al. 2019). This evidence concerns the gene FOXA1 and neoplasm.