MFGE8 and cardiac hypertrophy: In the aspect of cardiac fibrosis, Deng et al considered MFGE8 as an endogenous negative regulator of pathological cardiac hypertrophy, because after aortic coarctation in Mfge8‐knockout (Mfge8‐KO) mice, substantial hypertrophic enlargement in cardiomyocytes, systolic dysfunction, and myocardial fibrosis was observed.13