Since MSCs, besides others, reversed the NASH-induced decrease of Acaa1a, and PPAR signaling was among the significantly enriched pathways in the modules that showed an anti-correlation between +NASH-MSC vs. -NASH-MSC and +NASH+MSC vs. +NASH-MSC, we speculated that this pathway could play a role in the attenuation of pathological lipid storage in NASH livers. Here, PPARA is linked to metabolic dysfunction-associated steatohepatitis.