Urinary concentrations of NGAL and KIM-1 were significantly higher in patients with ATN without proliferative glomerulonephritis than in those with proliferative glomerulonephritis (NGAL: 146.2 [22.8-1195] vs 48.9 [13.1-287.9], P = 0.00324; KIM-1: 4884 [1568-6224] vs 1218 [820-2294], P=0.024). Here, LCN2 is linked to proliferative glomerulonephritis.