We identified four different mutations in LDLR and one in PCSK9. The majority of FH individuals carried mutation in LDLR (85.7% of index cases and 96.8% in total), which is similar to that reported in previous studies (Soutar and Naoumova, 2007; Sjouke et al., 2015; Khera et al., 2016). Here, PCSK9 is linked to familial hyperaldosteronism.