CFTR and pulmonary edema: Future research needs to establish whether as demonstrated in influenza A virus induced pulmonary edema it is, in addition to a dysfunction of the epithelial sodium channel possibly not a reduction but an activation of the CFTR (Wolk et al., 2008; Peteranderl et al., 2017), which is key in the pathogenesis e.g., via chloride secretion into the airway drawing water by osmosis with it.