Lupus-like syndrome is usually induced by drugs, more rarely by infections activating autoimmune response by transient positive autoantibodies, which can be associated with lupus-like symptoms such as malar rash, arthralgia, nephritis, and acute hepatitis.1 Our patient’s clinical presentation, including glomerular syndrome with significant proteinuria and hematuria, neuropsychiatric symptoms, lymphopenia, and positive ANA, fulfilled the 2012 Systemic Lupus International Collaborating Clinics revised classification criteria. Here, BTG3 is linked to systemic lupus erythematosus.