Recent studies have shown similarities between human and murine models of infectious disorders such as sepsis,(47) suggesting that there is some commonality in innate immune response to infection between species, including dysregulation of the murine CAMP homolog, cathelicidin‐related antimicrobial protein (CRAMP) in vitamin D‐deficient mice.(47) This study also highlighted another key facet of species homology in immune responses to vitamin D, namely the link between vitamin D deficiency and dysregulated inflammation in humans and mice. The gene discussed is CAMP; the disease is Sepsis.