TLX2 and Parkinson disease: The modulation of EAAT/NCX function may be an effective step forward in tackling the challenge of PD neurodegeneration, since it has the potential of simultaneously address both glutamate-mediated excitotoxicity (by favoring cell glutamate uptake) and the upstream energetic failure (by favoring glutamate metabolic use) that incite the progression of PD pathology.