IFNG and HIV infectious disease: Infection with VSV-G-NL4.3Δenv-eGFP HIV significantly increased production of CXCL10 by both HepG2 and AD38 cells in the presence of IFN-γ/P3CSK4 but there was no increase in CXCL10 following VSV-G-NL4.3Δenv-eGFP HIV infection alone (Fig 4).