To understand whether FABP4 plays a role in cardiomyocyte apoptosis associated with ischaemic condition of MI, we applied an in vitro hypoxia‐induced injury model, in which H9c2 cardiomyocytes were cultured deprived of serum in an anaerobic chamber, containing 5% CO2, 94% N2 and 1% O2, to induce hypoxia‐induced apoptosis.21 Here, FABP4 is linked to myocardial infarction.