To address whether TGFβ1 could serve as a trigger and modulator of structural plasticity in the stroke-denervated hemicord, we evaluated the expression pattern of TGFβ1 within the dorsal funiculus and gray matter at selected, early time points after stroke (2, 4, 7, and 28 dpi; Fig. 1A,B). The gene discussed is TGFB1; the disease is stroke disorder.