PTEN and familial dilated cardiomyopathy: MiR-132 activated the phosphateidylinositol 3-kinase/protein kinase (PI3K/Akt) signal transduction pathway via inhibiting phosphatase and tensin homolog (PTEN) expression, thus facilitating cardiocyte proliferation and attenuating apoptosis and cardiac fibrosis in rats with doxorubicin-induced dilated cardiomyopathy (DCM) [22].