Glioblastoma (GBM) cells have been shown to express Cx43, form homo‐cellular interactions with GBM cells, hetero‐cellular interactions with astrocytes, and demonstrate a positive correlation with Cx43 expression; glioma invasiveness and chemical or peptide blocking of gap junctions (GJ) inhibits migration and sensitizes GBM cells to ligand‐induced apoptosis. Here, GJA1 is linked to glioma.