This also likely explains why other disorders caused by ATP1A3 mutations, such as rapid onset dystonia parkinsonism (RDP) and relapsing encephalopathy with cerebellar ataxia (RECA) manifest marked bulbar findings of dysarthria, hypophonia, and dysphagia [10, 26]. The gene discussed is ATP1A3; the disease is Rapid-onset dystonia-parkinsonism.