In agreement with others findings (21), we found GDH hyperacetylated in T2D and hydroxyacyl-CoA dehydrogenase trifunctional multienzyme complex subunit α (HADHA) trended toward being hyperacetylated (P = 0.09) by T2D (Figure 6, A and B). The gene discussed is HADHA; the disease is type 2 diabetes mellitus.