Such genes include NF-κB, STAT1, major histocompatibility complex class I (MHC I), tumor necrosis factor (TNF), and beta interferon (IFN-β), potentially suggesting that neurons might be mounting typical cellular immune responses to infection and cytokine stimulation (7) and that TINs, the cells with intimate T. gondii contact, received more cytokine signaling and thus generated more robust cytokine responses. This evidence concerns the gene IFNB1 and infection.