Krober first provided PET/CT data for FDCS.[12] Smith reported that glucose transporter type 1, which is associated with glucose or 18F-FDG uptake, was overexpressed in malignant tumors.[13] Furthermore, Nakagawa reported the staining pattern for that glucose transporter type 1 and CD21 are markedly similar in lymphoid follicles, which is expressed exclusively in lymphoid follicles.[14] In the current case, we found that the tumor showed extremely avid FDG uptake, with an SUVmax value of 22.82. The gene discussed is CR2; the disease is neoplasm.