The bi-allelic deletion of the CDKN2A gene, further confirmed in a list of MM human cell lines including the least invasive MM34 (Meso 34), was found to be strongly associated with overexpression of IL34 and weakly with mutations of the NF2 gene (with no association with other genetic alterations in BAP1, LATS2 or TP53 genes) [16]. The gene discussed is TP53; the disease is Miyoshi myopathy.