Subsequently, literatures verification showed that inhibiting Angiotensin I converting enzyme (T1) could reduce mortality and the occurrence of severe left-ventricular dysfunction of Acute Myocardial Infarction (D1) patients [43] and Ginsenoside-Rg1 (C18) could enhance angiogenesis and ameliorates ventricular remodeling in a rat model of Acute Myocardial Infarction (D1) [41]. This evidence concerns the gene ACE and myocardial infarction.