MiR-9 could promote migration, invasion, and angiogenesis of endothelial progenitor cells via downregulating transient receptor potential melastatin 7 (TRPM7) and activating PI3K/Akt/autophagy pathway, which might facilitate thrombi recanalization in deep vein thrombosis (Zhou et al., 2020). The gene discussed is AKT1; the disease is deep vein thrombosis.