Since SARS-CoV-2 binds to ACE2 receptors to enter host cells, and intravenous infusion of ACE-inhibitors and angiotensin receptor blockers (ARBs) in experimental animal models increased the amount of ACE2 receptors in the cardiopulmonary circulation, it has been speculated that patients chronically taking these drugs may be at increased risk of worse outcomes from COVID-19 (68). Here, ACE is linked to COVID-19.