N-Glycosylation stabilizes CTHRC1 in oral squamous cell carcinoma (OSCC) specimens by reducing protein turnover rate, and CTHRC1 is positively feedback-regulated by the DPAGT1/canonical Wnt pathway, thereby activating noncanonical Wnt pathways to drive tumor cell migration and invasion [52]. Here, CTHRC1 is linked to neoplasm.