The methodology applied here might also be useful in the analysis of human proteins with N/Q biases, such as those linked to amytrophic lateral sclerosis or huntingtin from Huntington’s disease (An & Harrison, 2016; Monahan et al., 2018), or to other non-N/Q-biased prion-forming domains, such as in alpha-synuclein (Watts, 2019). This evidence concerns the gene SNCA and Huntington disease.