In support of the functional association of HMGA2 with cancers, pituitary adenomas develop in HMGA2‐transgenic mice and overexpression of HMGA2 induces neoplastic transformation in normal human lung cells whereas inhibiting HMGA2 expression suppresses the transformed phenotype in metastatic lung cancer cells (Di Cello et al., 2008; Fedele et al., 2002). This evidence concerns the gene HMGA2 and cancer.