The phenotype of the γδ T cells was characterized by high frequency of the marker CD16 but not the inhibitory receptor NKG2A (83.4 and 1.4%, respectively) on day 15 relative to that of HD (40.2% [17–52] and 33.7% [24.5–50] for CD16 and NKG2A, respectively) which persisted until death (Fig. 2d). The gene discussed is KLRC1; the disease is Huntington disease.