We then assessed the interaction of AGK and Hippo‐YAP1 pathway in gastric cancer cells and found that AGK overexpression was able to up‐regulate level of YAP1 and CTGF proteins, but lead to a down‐regulation of the level of LATS1/2 and p‐YAP without changing in MST1/2 levels (Figure 6A). The gene discussed is MST1; the disease is gastric cancer.