Conclusively, AB exerted antidiabetic, anti-obesity, antioxidative, and anti-inflammatory actions by reducing the expression of PPAR-γ and its modulators (C/EBP-ß, C/EBP-δ, and other C/EBP family members, Kruppel-like factor, and SREBP1C) Moreover, decreased expression level of transcriptomic factors such as C/EBPß (−0.41%), C/EBPα (−0.66%), Kruppel like factor (−0.83%), and SREBP1C (−0.24%) were also seen [125,133]. The gene discussed is CEBPA; the disease is obesity due to melanocortin 4 receptor deficiency.