Interestingly, DOX-elicited TEV could slightly stimulate the production of Bcl-xL in hypoxic C26 cancer cells by 25% (p = 0.0181, Figure 8C) compared to C26 hypoxic cells that received TEV, while RAW 264.7 recipient cells responded in a similar manner to either normoxic TEV and normoxic DOX-TEV compared to RAW 264.7 cells, which did not receive TEV (Figure 8E,F), thus showing a slight decrease by 20% (p = 0.0067 and p = 0.0091, respectively) of BAX levels in TEV recipient cells and a strong significant increase by 65% of Bcl-xL levels (p = 0.0043 and p = 0.0265, respectively). The gene discussed is BCL2L1; the disease is cancer.