Thus, 24 h of C26 cell DOX exposure under normoxic as well as hypoxic conditions moderately reduced (by 40–60%) the levels of a key transcription factor for tumour inflammation and angiogenesis, NF-κB p65 (p = 0.0276 and p = 0.0188, respectively) (Figure 6E and Figure 7E). The gene discussed is NFKB1; the disease is neoplasm.