Lastly, it was shown by Schilter et al. (2019) that administration of a LOXL2 inhibitor for 4-weeks, after left coronary arteries occlusion, resulted in an observed decreased myocardial fibrosis with improved cardiac output in a C57/BL6 mouse model [79]. The gene discussed is LOXL2; the disease is Myocardial fibrosis.