Although the fact that increased level or constitutive activation of c-Src was implied in the metastasis or carcinogenesis of colon, breast, lung, liver, pancreas, and prostate cancer cells [54,55,56], c-Src phosphorylation-dependent induction of hexokinase activity has been shown to stimulate the conversion of glucose to glucose-6-phosphate (G-6-P) associated with the carcinogenic and metastatic processes of cells [57]. The gene discussed is SRC; the disease is prostate carcinoma.