However, as we only evaluated the effect on proliferation and apoptosis, we cannot rule out that NDNF may be involved in MM through other functional mechanisms previously reported for this gene in different non-neoplastic contexts, such as cell adhesion, migration, or regulation of angiogenesis (Kuang et al. 2010; Ohashi et al. 2014). This evidence concerns the gene NDNF and Miyoshi myopathy.