In consistent with this finding, recent studies also show that overexpressing CEBP/β in young AD mice accelerates earlier onset of AD‐like pathologies and knockout of CEBP/β noticeably reduces neurofibrillary tangles in 3xTg mice (Wang, Liu, Chen, & Ye, 2018; Wang, Gong, et al., 2018). The gene discussed is CEBPA; the disease is Alzheimer disease.