Up to now, the Con A‐induced AIH mouse model was deemed to be a convenient and reliable animal model to mimic the pathological process of AIH in human, including increased production of proinflammatory cytokines, such as tumor necrosis factor‐α (TNF‐α) and interferon‐γ (IFN‐γ), necrosis of peripheral zonal liver cells and infiltration of cytotoxic cells [7]. Here, IFNG is linked to autoimmune hepatitis.