They identified higher levels of salivary IgA in CAD and atherosclerotic patients to MDA acetaldehyde–modified LDL and epitopes of pathogens such as gingipain A hemagglutinin domain of P. gingivalis (Rgp44), and A. actinomycetemcomitans HSP60 (AaHSP60) in comparison to non‐CAD patients reinforcing the importance of the role of bacterial HSP60, specifically oral pathogens, as risk factors for the progression of CVD.122. The gene discussed is CD79A; the disease is coronary artery disorder.