These results are in agreement with a previous study [29] reporting that iPSC-derived hNSCs are highly permissive to USUV infection, which leads to a reduction of cell viability of about 80% at day 4 p.i. and induction of caspase-3-dependent apoptosis at higher rate than after ZIKV infection. The gene discussed is CASP3; the disease is Zika virus infectious disease.